Fatigue in Medical Residents Leads to Reactivation of Herpes Virus Latency

Fatigue in Medical Residents Leads to Reactivation of Herpes Virus Latency

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The main objective of this study was to detect fatigue-induced clinical symptoms of immune suppression in medical residents.Samples were collected from the A Lazy Bailout Approach for Dual-Criticality Systems on Uniprocessor Platforms subjects at rest, following the first night (low-stress), and the last night (high-stress) of night float.Computerized reaction tests, Epworth Sleepiness Scale, and Wellness Profile questionnaires were used to quantify fatigue level.DNA of human herpes viruses HSV-1, VZV, EBV, as well as cortisol and melatonin concentrations, were measured in saliva.

Residents at the high-stress interval reported being sleepier compared to the rest interval.EBV DNA level increased significantly at both stress intervals, while VZV DNA level increased only at low-stress.DNA levels of HSV-1 decreased at low-stress but increased at high-stress.Combined assessment of the viral DNA showed significant effect Attosecond electron bunches from a nanofiber driven by Laguerre-Gaussian laser pulses of stress on herpes virus reactivation at both stress intervals.

Cortisol concentrations at both stress intervals were significantly higher than those at rest.